Here are three things to know about the finding.
• Prior to this discovery, researchers were aware sepsis occurred when molecules on the surface of certain bacteria infiltrated cells. This would result in an immune response that causes the cells to “self-destruct.”
• The newly identified gene, Gasdermin-D, is what they now believe deploys a protein that triggers cell death in the instance of bacterial invasion.
• In addition to enabling a better understanding of inflammatory diseases beyond sepsis, the researchers said the finding could potentially enable them to identify a mechanism to shut down sepsis before it becomes life-threatening.
More articles on infection control:
Medication errors occur in half of all surgeries: 7 things to know
California hospitals says endoscope was cleaned improperly for 7 years
5 essential components of a successful antibiotic stewardship program
At the Becker's 11th Annual IT + Revenue Cycle Conference: The Future of AI & Digital Health, taking place September 14–17 in Chicago, healthcare executives and digital leaders from across the country will come together to explore how AI, interoperability, cybersecurity, and revenue cycle innovation are transforming care delivery, strengthening financial performance, and driving the next era of digital health. Apply for complimentary registration now.